Scientists from the Centre for Allergy and Environment in Munich (ZAUM), the Helmholtz Zentrum München and the Technische Universität München have completed a study examining patients with both eczema and psoriasis.
The National Eczema Association (NEA) research program offers grants to established and new investigators in the field. The 2011 research areas of emphasis are: Eczema prevention, Alternative therapies, Itch, Co-morbidities.
Researchers at Yale and Johns Hopkins have discovered that BAM8-22, a signaling molecule found in the skin, triggers itch in humans via a pathway that does not involve histamine.
A University of Missouri forestry professor has found that a species of cedar tree carries an antibiotic that appears to be effective against the bacterial infection methicillin-resistant Staphylococcus aureus (MRSA).
According to a new study presented at the annual congress of the European Academy for Dermatology and Venereology, compliance with topical dermatologic therapies is worse than most physicians realize.
NEA Researchers Dr. Anna De Benedetto and Dr. Lisa A. Beck led a group of scientists in studying the function of one of the two main skin barriers, the tight junction (TJ) to determine the role of claudin-1, a protein component of TJ, in Atopic Dermatitis.
In this study, Tatyana E. Shaw, Gabriel P. Currie, Caroline W. Koudelka, and Eric L. Simpson of the Oregon Health & Sciences University, Department of Dermatology, calculated estimates of pediatric eczema population throughout the United States.
A new study in human cells has singled out a molecule that specifically directs immune cells to develop the capability to produce an allergic response. The signaling molecule, called thymic stromal lymphopoietin (TSLP), is key to the development of allergic diseases such as asthma, atopic dermatitis (eczema), and food allergy.
Chronic itch is an often difficult and sometimes debilitating symptom of many skin diseases and other disorders. Researchers have been trying to determine for decades if there are separate neuronal pathways for pain and itch.
